WorldCat Identities

Moran, Christopher J.

Overview
Works: 18 works in 21 publications in 1 language and 247 library holdings
Genres: History 
Roles: Author, Contributor, Other
Classifications: KFN5210.Z9, 364.162
Publication Timeline
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Most widely held works by Christopher J Moran
Preventing embezzlement by Christopher J Moran( Book )

3 editions published in 1982 in English and held by 154 WorldCat member libraries worldwide

Preventing embezzlement by Christopher J Moran( Book )

2 editions published in 1978 in English and held by 65 WorldCat member libraries worldwide

Neuroradiology since the advent of computed tomography by Christopher J Moran( Book )

1 edition published in 1980 in English and held by 6 WorldCat member libraries worldwide

Preventing embezzlement by Christopher J Moran( Book )

1 edition published in 1978 in English and held by 5 WorldCat member libraries worldwide

Acute Stroke Intervention( )

1 edition published in 2003 in English and held by 2 WorldCat member libraries worldwide

Endovascular Management of Cerebral Arteriovenous Malformations( )

1 edition published in 2003 in English and held by 2 WorldCat member libraries worldwide

Neurointerventional Materials( )

1 edition published in 2003 in English and held by 2 WorldCat member libraries worldwide

Endovascular Treatment of Intracranial Aneurysms( )

1 edition published in 2003 in English and held by 2 WorldCat member libraries worldwide

Preventing embezzlement by Christopher J Moran( Book )

1 edition published in 1973 in English and held by 2 WorldCat member libraries worldwide

Very Early-onset Inflammatory Bowel Disease( )

1 edition published in 2015 in English and held by 1 WorldCat member library worldwide

Abstract : Abstract: The pathogenesis of pediatric inflammatory bowel disease (IBD) is only partially understood. Strong evidence implicates a strong genetic component including high monozygotic twin concordance and familial disease phenotype concordance rates. Genome-wide association studies have identified associations between>160 genetic loci and the risk for developing IBD. The roles of implicated genes are largely immune-mediated, although other functions include cellular migration, oxidative stress, and carbohydrate metabolism. Additionally, growing literature describes monogenic causes of IBD that frequently present as infantile or very early-onset IBD. The interplay between IBD risk single nucleotide polymorphisms and rare genetic variants has yet to be determined. Studying patients with very early-onset IBD may elicit genetic factors that could be applied to broader populations of IBD. This review describes what is known about the genetic causes of very early-onset IBD and genetic strategies that may unravel more of the genetic causes of IBD. Abstract : Article first published online 26 February 2015
Direct puncture Onyx embolization of a large calvarial metastasis with intracranial extension: Case report( )

in English and held by 1 WorldCat member library worldwide

We report a case of renal cell carcinoma (RCC) metastasis to the calvarium and describe a strategy for percutaneous embolization of hypervascular calvarial tumors with intracranial extension. An elderly patient with history of RCC presented with left-sided weakness. Imaging studies showed a large right frontoparietal calvarial mass with intra- and extracranial extension. The tumor was devascularized by direct puncture tumor embolization using Onyx 18, allowing subsequent operative resection without significant blood loss or the need for flap reconstruction of the scalp. Compared to more common endovascular approaches, direct-needle puncture embolization of transcalvarial masses may offer lower risk of injury to scalp vessels and underlying brain parenchyma
Very Early-onset Inflammatory Bowel Disease( )

1 edition published in 2015 in English and held by 1 WorldCat member library worldwide

Abstract : Abstract: The pathogenesis of pediatric inflammatory bowel disease (IBD) is only partially understood. Strong evidence implicates a strong genetic component including high monozygotic twin concordance and familial disease phenotype concordance rates. Genome-wide association studies have identified associations between>160 genetic loci and the risk for developing IBD. The roles of implicated genes are largely immune-mediated, although other functions include cellular migration, oxidative stress, and carbohydrate metabolism. Additionally, growing literature describes monogenic causes of IBD that frequently present as infantile or very early-onset IBD. The interplay between IBD risk single nucleotide polymorphisms and rare genetic variants has yet to be determined. Studying patients with very early-onset IBD may elicit genetic factors that could be applied to broader populations of IBD. This review describes what is known about the genetic causes of very early-onset IBD and genetic strategies that may unravel more of the genetic causes of IBD. Abstract : Article first published online 26 February 2015
Las Vegas : 1928 to 1931 the years of transition by Christopher J Moran( Book )

1 edition published in 1975 in English and held by 1 WorldCat member library worldwide

Authors' Response( )

1 edition published in 2016 in English and held by 1 WorldCat member library worldwide

Do mountain pine beetle outbreaks change the probability of active crown fire in lodgepole pine forests? Comment by Christopher J Moran( )

1 edition published in 2012 in English and held by 1 WorldCat member library worldwide

CRANIAL DURAL ARTERIOVENOUS FISTULAE( )

1 edition published in 2009 in English and held by 1 WorldCat member library worldwide

Abstract OBJECTIVE Cranial dural arteriovenous fistulae (dAVF) with cortical venous drainage (CVD) (Borden Types 2 and 3) are reported to carry a 15% annual risk of intracranial hemorrhage (ICH) or nonhemorrhagic neurological deficit (NHND). The purpose of this study was to compare the clinical course of Type 2 and 3 dAVFs that present with ICH or NHND with those that do not. METHODS Twenty-eight patients with Type 2 or 3 dAVFs were retrospectively evaluated. CVD was classified as asymptomatic (aCVD) if patients presented incidentally or with pulsatile tinnitus or orbital phenomena. CVD was classified as symptomatic (sCVD) if patients presented with ICH or NHND. Occurrence of new ICH or new or worsening NHND between diagnosis and disconnection of CVD or last follow-up (if not disconnected) was noted. Overall frequency of events was compared using Fisher's exact test. Cumulative, event-free survival was compared using Kaplan-Meier analysis with log-rank testing. RESULTS Of 17 patients with aCVD, 1 (5.9%) developed ICH and none experienced NHND or death during the median 31.4-month follow-up period. Of 11 patients with sCVD, 2 (18.2%) developed ICH and 3 (27.3%) experienced new or worsened NHND over the median 9.7-month follow-up period. One of these patients subsequently died. Overall frequency of ICH or NHND was significantly lower in patients with aCVD versus sCVD (P = 0.022). Respective annual event rates were 1.4 versus 19.0%. aCVD patients had significantly higher cumulative event-free survival (P = 0.0016). CONCLUSION Cranial dAVFs with aCVD may have a less aggressive clinical course than those with sCVD
Estate probate & administration( Recording )

1 edition published in 1974 in English and held by 0 WorldCat member libraries worldwide

Spatial data mining for enhanced soil map modelling by Christopher J Moran( )

1 edition published in 2002 in English and held by 0 WorldCat member libraries worldwide

 
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Audience Level
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Audience Level
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  Kids General Special  
Audience level: 0.61 (from 0.07 for Preventing ... to 1.00 for Spatial da ...)

Languages
English (21)