WorldCat Identities

Robbins, Jeffrey

Overview
Works: 14 works in 72 publications in 7 languages and 2,444 library holdings
Genres: Anecdotes  Biography  Miscellanea  Popular works 
Roles: Editor, Other, Author
Publication Timeline
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Most widely held works by Jeffrey Robbins
The pleasure of finding things out : the best short works of Richard P. Feynman by Richard P Feynman( Book )

39 editions published between 1999 and 2007 in English and Dutch and held by 2,138 WorldCat member libraries worldwide

A treasury of short writings by twentieth-century American physicist Richard Feynman, including interviews, speeches, lectures, and printed articles, in which he shares details of his private life, his work on the atomic bomb, and his conclusions on the explosion of the space shuttle Challenger
The pleasure of finding things out by Richard P Feynman( Recording )

10 editions published in 2013 in English and held by 147 WorldCat member libraries worldwide

The Pleasure of Finding Things Out is a magnificent treasury of the best short works of Richard P. Feynman, from interviews and speeches to lectures and printed articles. A sweeping, wide-ranging collection, it presents an intimate and fascinating view of a life in science-a life like no other. From his ruminations on science in our culture to his Nobel Prize acceptance speech, this book will delight anyone interested in the world of ideas. "From the irregular trivia of ordinary life mixed with a bit of scientific doodling and failure to the intense dramatic concentration as one closes in on the truth and the final elation (plus, with gradually decreasing frequency, the sudden sharp pangs of doubt)-that is how science is done."--Richard P. Feynman to James D. Watson
Es ist so einfach : vom Vergnügen, Dinge zu entdecken by Richard P Feynman( Book )

6 editions published between 2001 and 2011 in German and held by 10 WorldCat member libraries worldwide

Il piacere di scoprire by Richard P Feynman( Book )

2 editions published between 2002 and 2004 in Italian and held by 4 WorldCat member libraries worldwide

Il bambino e l'integrazione sensoriale : le sfide nascoste della sensorialità by A. Jean Ayres( Book )

1 edition published in 2012 in Italian and held by 3 WorldCat member libraries worldwide

The preparation and characterization of myosin complementary DNA by Jeffrey Robbins( Book )

2 editions published in 1976 in English and held by 2 WorldCat member libraries worldwide

Cardioskeletal myopathies in children and young adults by John Lynn Jefferies( Book )

3 editions published in 2017 in English and held by 1 WorldCat member library worldwide

<> by Richard P Feynman( Book )

1 edition published in 2007 in Hebrew and held by 1 WorldCat member library worldwide

MMI‐0100 Inhibits Cardiac Fibrosis in a Mouse Model Overexpressing Cardiac Myosin Binding Protein C( )

1 edition published in 2017 in English and held by 1 WorldCat member library worldwide

Abstract : Background: Cardiac stress can trigger production of a 40‐kDa peptide fragment derived from the amino terminus of the cardiac myosin‐binding protein C. Cardiac stress, as well as cMyBP‐C mutations, can trigger production of 1 such truncated protein fragment, a 40‐kDa peptide fragment derived from the amino terminus of cMyBP‐C. Genetic expression of this 40‐kDa fragment in mouse cardiomyocytes (cMyBP‐C 40k ) leads to cardiac disease, fibrosis, and death within the first year. Fibrosis can occur in many cardiovascular diseases, and mitogen‐activated protein kinase––activated protein kinase‐2 signaling has been implicated in a variety of fibrotic processes. Recent studies demonstrated that mitogen‐activated protein kinase––activated protein kinase‐2 inhibition using the cell‐permeant peptide inhibitor MMI‐0100 is protective in the setting of acute myocardial infarction. We hypothesized that MMI‐0100 might also be protective in a chronic model of fibrosis, produced as a result of cMyBP‐C 40k cardiomyocyte expression. Methods and Results: Nontransgenic and cMyBP‐C 40k inducible transgenic mice were given MMI‐0100 or PBS daily for 30 weeks. In control groups, long‐term MMI‐0100 was benign, with no measurable effects on cardiac anatomy, function, cell viability, hypertrophy, or probability of survival. In the inducible transgenic group, MMI‐0100 treatment reduced cardiac fibrosis, decreased cardiac hypertrophy, and prolonged survival. Conclusions: Pharmaceutical inhibition of mitogen‐activated protein kinase––activated protein kinase‐2 signaling via MMI‐0100 treatment is beneficial in the context of fibrotic cMyBPC 40k disease
Keşfetme hazzı : Richard P. Feynman'ın en iyi kısa çalışmaları by Richard P Feynman( Book )

1 edition published in 2002 in Turkish and held by 1 WorldCat member library worldwide

Ē chara tēs anakalypsēs : ta kalytera mikra erga tou R.P. Feynman by Richard P Feynman( Book )

1 edition published in 2005 in Greek, Modern and held by 1 WorldCat member library worldwide

Activation of Autophagy Ameliorates Cardiomyopathy in Mybpc3-Targeted Knockin Mice( )

1 edition published in 2017 in English and held by 1 WorldCat member library worldwide

Abstract : Background: Alterations in autophagy have been reported in hypertrophic cardiomyopathy (HCM) caused by Danon disease, Vici syndrome, or LEOPARD syndrome, but not in HCM caused by mutations in genes encoding sarcomeric proteins, which account for most of HCM cases. MYBPC3, encoding cMyBP-C (cardiac myosin-binding protein C), is the most frequently mutated HCM gene. Methods and Results: We evaluated autophagy in patients with HCM carrying MYBPC3 mutations and in a Mybpc3 -targeted knockin HCM mouse model, as well as the effect of autophagy modulators on the development of cardiomyopathy in knockin mice. Microtubule-associated protein 1 light chain 3 (LC3)-II protein levels were higher in HCM septal myectomies than in nonfailing control hearts and in 60-week-old knockin than in wild-type mouse hearts. In contrast to wild-type, autophagic flux was blunted and associated with accumulation of residual bodies and glycogen in hearts of 60-week-old knockin mice. We found that Akt-mTORC1 (mammalian target of rapamycin complex 1) signaling was increased, and treatment with 2.24 mg/kg·d rapamycin or 40% caloric restriction for 9 weeks partially rescued cardiomyopathy or heart failure and restored autophagic flux in knockin mice. Conclusions: Altogether, we found that (1) autophagy is altered in patients with HCM carrying MYBPC3 mutations, (2) autophagy is impaired in Mybpc3 -targeted knockin mice, and (3) activation of autophagy ameliorated the cardiac disease phenotype in this mouse model. We propose that activation of autophagy might be an attractive option alone or in combination with another therapy to rescue HCM caused by MYBPC3 mutations. Abstract : Supplemental Digital Content is available in the text
Il piacere di scropire by Richard P Feynman( Book )

1 edition published in 2002 in Italian and held by 1 WorldCat member library worldwide

The pleasure of finding things out : the best short works of Richard P. Feynman ; edited by Jeffrey Robbins ; foreword by Freeman Dyson by Richard P Feynman( )

1 edition published in 1999 in English and held by 0 WorldCat member libraries worldwide

 
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The pleasure of finding things out : the best short works of Richard P. Feynman
Alternative Names
רובינס, ג׳פרי

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