WorldCat Identities

Beurton, Alexandra

Overview
Works: 8 works in 8 publications in 2 languages and 13 library holdings
Roles: Other, Author
Publication Timeline
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Most widely held works by Alexandra Beurton
Erratum to: Cardiac dysfunction induced by weaning from mechanical ventilation: incidence, risk factors, and effects of fluid removal by Jinglun Liu( )

1 edition published in 2017 in English and held by 2 WorldCat member libraries worldwide

Carotid and femoral Doppler do not allow the assessment of passive leg raising effects by Valentina Girotto( )

1 edition published in 2018 in English and held by 2 WorldCat member libraries worldwide

Transpulmonary thermodilution detects rapid and reversible increases in lung water induced by positive end-expiratory pressure in acute respiratory distress syndrome by Francesco Gavelli( )

1 edition published in 2020 in English and held by 2 WorldCat member libraries worldwide

Cardiac dysfunction induced by weaning from mechanical ventilation: incidence, risk factors, and effects of fluid removal by Jinglun Liu( )

1 edition published in 2016 in English and held by 2 WorldCat member libraries worldwide

The effects of passive leg raising may be detected by the plethysmographic oxygen saturation signal in critically ill patients by Alexandra Beurton( )

1 edition published in 2019 in English and held by 2 WorldCat member libraries worldwide

Une présentation multikystique d'adénocarcinome lépidique mucineux by Alexandra Beurton( Book )

1 edition published in 2015 in French and held by 1 WorldCat member library worldwide

INTRODUCTION: The pneumonic form is very characteristic of the extended lepidic lung adenocarcinoma. However, the diagnosis and treatment in this form may be delayed by radiological atypia and severity of hypoxemia. CASE REPORT: A 48 year old female, non-smoker, asthmatic since the age of 20 years, was hospitalized for a diffuse infiltrative pneumonia complicated with a severe respiratory failure. The history included chronic cough, gradually increasing dyspnea on exertion lasting for 14 months and the onset of hemoptysis low abundance associated with arthralgia since a month. She had no professional or domestic risk factor. Chest CT scan revealed bilateral alveolar condensations, ground glass areas with thickened septa creating a crazy paving pattern, and numerous large cysts of various sizes and locations, often with irregular thin wall. The microbiological and immunological tests were negative. Despite an early invasive ventilation and ECMO venovenous established before a refractory hypoxemia and a broad-spectrum empiric antibiotic therapy, evolution was fatal after 39 days of intensive care. The autopsy revealed lesions consistent with mucinous adenocarcinoma lepidique not expressing EGFR mutation and KRAS associated with pulmonary suppuration. CONCLUSION: Mucinous adenocarcinoma lepidique is an alternative diagnosis of pneumonic condensation associated with multiple cysts
Beneficial Effects of Norepinephrine Alone on Cardiovascular Function and Tissue Oxygenation in a Pig Model of Cardiogenic Shock( )

1 edition published in 2016 in English and held by 1 WorldCat member library worldwide

Abstract : Supplemental Digital Content is available in the text Abstract : ABSTRACT: Introduction: The present study was developed to investigate the effects of norepinephrine alone on hemodynamics and intrinsic cardiac function in a pig model of cardiogenic shock mimicking the clinical setting. Methods: Cardiogenic shock was induced by 1-h ligation of the left anterior descending (LAD) artery followed by reperfusion. Pigs were monitored with a Swan-Ganz catheter, a transpulmonary thermodilution catheter, and a conductance catheter placed in the left ventricle for pressure-loop measurements. Measurements were performed before LAD occlusion, 1 h after LAD occlusion, and 4 h after myocardial reperfusion. Results: Myocardial infarction and reperfusion was followed by cardiogenic shock characterized by a significant increase in heart rate and significant decreases in mean arterial pressure (MAP), mixed venous oxygen saturation (SVO2), left ventricular end-diastolic pressure (LVEDP), prerecruitable stroke work (PRSW), and cardiac power index (CPI). Lactate levels were significantly increased. The systemic vascular resistance index (SVRI) and global end-diastolic volume index (GEDVI) remained unchanged. When compared with the control group (n = 6), norepinephrine infusion (n = 6) was associated with no changes in heart rate, a significant increase in MAP, SVO2, left ventricular ejection fraction, pressure development during isovolumic contraction, SVRI, and CPI and a decrease in lactate level. Cardiac index tended to increase (P = 0.059), whereas PRSW did not change in the norepinephrine group. LVEDP and GEDVI remained unchanged. Conclusions: Norepinephrine alone is able to improve hemodynamics, cardiac function, and tissue oxygenation in a pig model of ischemic cardiogenic shock
Validation du dispositif de thermodilution transpulmonaire sur un modèle de choc cardiogénique ischémique porcin by Alexandra Beurton( )

1 edition published in 2015 in French and held by 1 WorldCat member library worldwide

Le monitorage hémodynamique par système PiCCO® (Pulsion Medical System AG) est validé dans plusieurs situations cliniques. Il existe peu de données validant son utilisation au cours du choc cardiogénique. La noradrénaline est l'amine de choix dans le choc cardiogénique non ischémique selon les recommandations récentes RFE SRLF 2014. Cependant, aucune étude ne montre son intêret dans le choc cardiogénique ischémique. OBJECTIFS : Comparer les données fournies par le cathéter PiCCO® à celles de méthodes de référence d'évaluation hémodynamique (cathéters de Swan Ganz et Millar) dans un modèle de choc cardiogénique ischémique porcin réanimé. Evaluer l'effet de la noradrénaline dans le choc cardiogénique ischémique.METHODES : Il s'agit d'un modèle de choc cardiogénique par ligature de l'artère interventriculaire antérieure. L'ensemble des données hémodynamiques était recueilli avant et au moment du choc, puis toutes les heures pendant quatre heures. Douze cochons étaient répartis en deux groupes, un groupe sham (n = 6) s ans traitement et un groupe traité (n = 6) par noradrénaline pour maintenir une PAm supérieure ou égale à 65mmHg. RESULTATS : Les paramètres initiaux montraient, pour le groupe non traité et le groupe traité respectivement, une PAm de 90,7mmHg +/- 8,1 vers us 100,8mmHg +/- 20,7 ; IC PiCCO ® moy en de 3L/min/m² +/- 0,7 versus 2,8 +/- 0,7 ; une SvO2 de 65% dans les deux groupes. Au moment du choc, on observait une PAm de 68,8mmHg +/- 15 versus 53mmHg +/- 19,2 ; un IC PiCCO ® moyen de 1,7L/min/m² +/- 0,4 versus 1,6L/min/m² +/- 0,6 ; une SvO2 de 38,7% +/- 9,5 versus 39,7% +/- 15,4. Ilexistait une corrélation significative entre les IC du système PiCCO ® et le cathéter de Millar r = 0,358 [IC 95% 0,119-0,558] p = 0,004, entre les FE r = 0,299 [IC 95% 0,051-0,512] p = 0,019, entre la PTDVG et la PAPO r = 0,324 [IC 95% 0,050-0,552] p = 0,021. La noradrénaline permet une augmentation de la PAm de 26mmHg +/ - 14,3 p=0,033, de la FEG PiCCO® de 6,4% +/- 2,4 p=0,024, de la SvO2 de 14,1% +/- 5,5 p=0,033, de la FEVG donnée par le cathéter Millar de 8,7% +/- 4,1 p=0,043, du dP /dt max de 1345mmHg/s +/-465 p=0,024 ; sans augmentation significative de la FC. CONCLUSION : Dans notre modèle, notre étude permet de montrer que les corrélations sont significatives mais faibles entre les trois outils hémodynamiques pour les principaux paramètres utilisés en pratique clinique. En effet, en terme de valeur absolue, la significativité est peu importante mais l'évolution est significative, et cela est primordial pour le suivi des thérapeutiques instaurées.La noradrénaline augmente de manière signific ative la FE, la PAm, la SvO2, le dP /dt max sans augmenter la FC, et s'avère être le traitement de choix dans le choc cardiogénique ischémique
 
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