WorldCat Identities

Pecchi, Emilie

Overview
Works: 4 works in 5 publications in 2 languages and 9 library holdings
Roles: Author, Contributor, Other
Publication Timeline
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Most widely held works by Emilie Pecchi
Mécanismes moléculaires de l'anorexie inflammatoire : rôle de la microsomal Prostaglandin E Synthase (mPGES)-1 by Emilie Pecchi( Book )

2 editions published in 2008 in French and held by 3 WorldCat member libraries worldwide

Infection or inflammation trigger a set of physiological changes (anorexia, fever, ...) known as the acute phase reaction. During this work, we analyzed the possible involvement of a terminal prostaglandin E2 synthase, the mPGES-1, in this pathological state. We show that this enzyme, inducible in inflammatory conditions, is required for central nervous system activation and for anorexia during acute inflammation. Moreover its invalidation abolished the symptoms of the anorexia-cachexia syndrome (chronic anorexia and dramatic weight loss) induced by subcutaneous tumour growth. Altogether, our results indicate that mPGES-1 inhibition could represent a therapeutic strategy to treat acute phase reaction symptoms and anorexia-cachexia syndrome observed in numerous chronic diseases with inflammatory component
MALDI-TOF MS as an innovative tool for detection of Plasmodium parasites in Anopheles mosquitoes by Maureen Laroche( )

1 edition published in 2017 in English and held by 2 WorldCat member libraries worldwide

Central nesfatin-1-expressing neurons are sensitive to peripheral inflammatory stimulus by Marion S Bonnet( )

1 edition published in 2009 in English and held by 2 WorldCat member libraries worldwide

Induction of nerve growth factor expression and release by mechanical and inflammatory stimuli in chondrocytes: possible involvement in osteoarthritis pain by Emilie Pecchi( )

1 edition published in 2014 in English and held by 2 WorldCat member libraries worldwide

 
Audience Level
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Audience Level
1
  General Special  
Audience level: 0.00 (from 0.00 for Mécanisme ... to 0.00 for Mécanisme ...)

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