WorldCat Identities

Hourde, Christophe

Overview
Works: 7 works in 8 publications in 2 languages and 12 library holdings
Roles: Other, Author, Contributor, Opponent
Publication Timeline
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Most widely held works by Christophe Hourde
Viral-mediated expression of desmin mutants to create mouse models of myofibrillar myopathy by Pierre Joanne( )

1 edition published in 2013 in English and held by 2 WorldCat member libraries worldwide

Necroptosis mediates myofibre death in dystrophin-deficient mice by Jennifer E Morgan( )

1 edition published in 2018 in English and held by 2 WorldCat member libraries worldwide

Étude de la coordination des phénotypes contractile et métabolique du muscle strié squelettique à travers des modèles animaux by Christophe Hourdé( Book )

2 editions published in 2006 in French and held by 2 WorldCat member libraries worldwide

There exists little information on the functional recovery and the relationships between functional parameters, biochemistry and the structural aspect of the recovery. This is why, we have combined physiological, biochemical and structural evaluations of the regenerating skeletal muscles in various models of lesions (venom, ischemia and venom). Although the biochemical criteria suggest a complete recovery, it is not the case from the functional xiewpoint. Our work thus shows that the examination of the contractile properties after a lesion is the most relevant method of evaluation to appreciate the degree of muscle recovery. It was recently recommended to analyze the molecular regulations by group of functional genes or modules (contractile proteins, mitochondria, ionic, fatty acid or glucose transport, etc). With the aim of progressing in understanding the coordinated modulations governing muscle plasticy, we undertook to characterize changes in gene modules corresponding to energy metabolism and to isoforms of contractile proteins. We chose specific markers of these moduls to study animal models of neuromuscular modifications corresponding to either hypodynamy (microgravity simulated by suspension) or hyperdynamy (training), in slow muscles. For that purpose, we developed micromethods allowing to obtain data on expression changes of many markers, starting from only one muscle. We thus developed an analysis of coordinated modifications, bringing a global perspective, although more restricted than the proteomic or genomic approaches developed these past years
Inflammatory Response 24 h Post-Exercise in Youth with Juvenile Idiopathic Arthritis( )

1 edition published in 2018 in English and held by 2 WorldCat member libraries worldwide

Publisher Correction: Necroptosis mediates myofibre death in dystrophin-deficient mice by Jennifer E Morgan( )

1 edition published in 2018 in English and held by 2 WorldCat member libraries worldwide

Adaptations cellulaires et moléculaires de la réponse inflammatoire à l'activité physique chez les enfants atteints d'arthrite juvénile idiopathique by Emmanuelle Gallion-Rochette( )

1 edition published in 2018 in French and held by 1 WorldCat member library worldwide

Étude de la coordination des phénotypes contractile et métabolique du muscle strié squelettique à travers des modèles animaux by Christophe Hourdé( )

1 edition published in 2006 in French and held by 1 WorldCat member library worldwide

There exists little information on the functional recovery and the relationships between functional parameters, biochemistry and the structural aspect of the recovery. This is why, we have combined physiological, biochemical and structural evaluations of the regenerating skeletal muscles in various models of lesions (venom, ischemia and venom). Although the biochemical criteria suggest a complete recovery, it is not the case from the functional xiewpoint. Our work thus shows that the examination of the contractile properties after a lesion is the most relevant method of evaluation to appreciate the degree of muscle recovery. It was recently recommended to analyze the molecular regulations by group of functional genes or modules (contractile proteins, mitochondria, ionic, fatty acid or glucose transport, etc). With the aim of progressing in understanding the coordinated modulations governing muscle plasticy, we undertook to characterize changes in gene modules corresponding to energy metabolism and to isoforms of contractile proteins. We chose specific markers of these moduls to study animal models of neuromuscular modifications corresponding to either hypodynamy (microgravity simulated by suspension) or hyperdynamy (training), in slow muscles. For that purpose, we developed micromethods allowing to obtain data on expression changes of many markers, starting from only one muscle. We thus developed an analysis of coordinated modifications, bringing a global perspective, although more restricted than the proteomic or genomic approaches developed these past years
 
Audience Level
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Audience Level
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  General Special  
Audience level: 0.95 (from 0.86 for Adaptation ... to 0.97 for Étude de ...)

Alternative Names
Christophe Hourdé researcher

Christophe Hourdé wetenschapper

Languages